Emory University researchers find amyloid beta deposits in Alzheimer's patients' brains act as a scaffold for other proteins potentially causing brain cell damage.

Emory University researchers challenge traditional Alzheimer's theories, finding that amyloid beta deposits in patients' brains act as a scaffold for other proteins potentially causing brain cell damage. Over 20 proteins co-accumulate with amyloid beta in human brains with Alzheimer's and mice, suggesting these additional proteins may play a role in brain damage, leading to potential new therapies and treatment targets for Alzheimer's and other diseases.

August 12, 2024
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