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A Johns Hopkins study links a brain protein, CSE, to Alzheimer’s protection in mice, pointing to a new treatment target.
A Johns Hopkins-led study funded by the NIH finds that a protein called CSE, which produces hydrogen sulfide in the brain, may be a promising target for Alzheimer’s treatments.
Mice lacking CSE developed memory issues, reduced neuron growth, and blood vessel damage—features linked to Alzheimer’s—suggesting the protein helps protect brain health.
While human applications remain uncertain, the findings highlight a new biological pathway for potential therapies.
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Un estudio de Johns Hopkins vincula una proteína cerebral, CSE, a la protección del Alzheimer en ratones, apuntando a un nuevo objetivo de tratamiento.